Salt
Serotonin
Skin
Soy
Soy and Infants
Soy and Sex
Sugar: (also see Carbohydrates)
Sweeteners
Syndrome X
Salt:
Stomach acid requires chloride from salt. Reference: Body Fluids
and Electrolytes, pgs: 20-22., Norma J. Weldy, Mosby-Year
Book, November, 1991, ISBN: 0801654017
Serotonin:
Carbohydrates (sugar) increase the levels of both beta-endorphin and
serotonin in the brain. These brain chemicals create feelings of calmness
and even euphoria. The trouble with this is the more sugar eaten, the
more the brain has to compensate for this “excess” by shutting
down some of its beta-endorphin and serotonin receptors. This creates
a sugar sensitive system, where after the sugar high, comes a sugar
low, which causes negative feelings, like depression, and cravings for
more carbohydrates. A person who is sugar sensitive (addicted to carbs)
has less ability to say “no” due to the effect of this chemical
imbalance in the brain. Brain Serotonin, Carbohydrate-craving, obesity
and depression. Reference: Wurtman RJ, Wurtman JJ. Department of
Brain and Cognitive Sciences, Clinical Research Center Massachussetts
Institute of Technology, Cambridge 02139, USA.
Skin:
When EFA oils are applied to the skin “…there was penetration
and incorporation of their (EFAs) constituent fatty acids, into structural
lipid of the epidermis. As the oils were triglyceride forms of the fatty
acids, it is clear that they were actually metabolized by the skin…”
“…the applied oil is metabolized by the skin and linolenic
acid is incorporated into structural lipid.” What this means is
that used topically, the skin actually metabolizes EFAs. Reference:
The Journal of Investigative Dermatology, 64:228-234, 1975, Vol. 64,
No. 4, Colin Prottey, PhD., Peter J. Hartop, S.S.c., and Martin Press,
F.R.C.P., The William and Wilkins Company.
“EFA's maintain youthful, radiant skin by preserving the lipid
barriers between the skin's upper and lower layers. They are also actively
involved in replenishing collagen and elastin in the dermis.”
Reference: 1 Cunnane SC. In: Flaxseed in Human Nutrition. Cunnane
SC and Thompson LU, eds. Champaign, IL: AOCS Press, 1995, p. 123.
“Essential Fatty Acids help hydrate and increase the skin’s
moisture content.” Reference: Pepper, G. Nutrients for Healthy
Skin, Hair & Nails. Metabolism.com Web site. 2003. Available at:
http://www.metabolism.com/news/2001-12-04/. Accessed April 11, 2003.
(With a deficiency in EFAs) Skin becomes rough and inflamed with a poor
wound healing capacity. Reference: Wright S. Essential fatty
acids in clinical dermatology. J Nutr Med 1:301-313, 1990.
“After cutaneous (topical) application of sunflower-seed oil,
which is rich in linolenic acid (omega 3), to their (3 patients previously
shown to be EFA deficient and suffering scaly lesions) right forearms
for 2 weeks, the level of linolenic acid in their epidermal (skin) lecithin
was markedly increased, the rate of transepidermal water loss was significantly
lowered, and the scaly lesions disappeared. No such changes were seen
in their left forearms after cutaneous application of olive oil (rich
in omega 9)…” Reference: The Journal of Investigative
Dermatology, 64:228-234, 1975, Vol. 64, No. 4, Colin Prottey, PhD.,
Peter J. Hartop, S.S.c., and Martin Press, F.R.C.P., The William and
Wilkins Company.
Just about any type of dermatitis can favorably respond
to EFA supplementation. Topical application along with oral supplementation
may speed the effectiveness of EFAs. Eczema, psoriasis and acne all
have been shown to respond to EFA supplementation. Reference: Wright
S, Burton JL. Evening primrose seed oil improves atopic eczema. Lancet
ii:1120-1122, 1982.
Bittiner SB, Tucker WFG, Cartwright I, Bleehen SS. A double blind randomized,
placebo-controlled trial of fish oil in psoriasis. Reference: Lancet
i:378-380, 1988.
Reference: Downin DT, Stweart ME, Wertz PW, Strauss JS. Essential
fatty acids and acne. J Am Acad Dermat 14:221-4, 1986.
Skin disorders and problems such as atopic dermatitis or eczema, dry
skin, psoriasis, increased transepidermal water loss (TEWL) and impaired
epidermal barrier function is associated with deficiencies in GLA (Omega
6). Both oral and topical administration of GLA has been effective in
reducing these as well as reducing redness and erythema due to UV radiation
and improving healing of wounds. Reference: Melnick B, Plewig G.
Atopic dermatitis and disturbances in essential fatty acid and prostaglandin
E metabolism. J Amer Acad Dermatol 1991;25:859
Reference: Campbell KL. Fatty acid supplementation and skin disease.
Adv Clin Derm 1990;20(6):1475-1486.
The topical administration of borage oil has been shown to be very effective
in preventing and treating inflammatory conditions and skin disorders
such as eczema and dermatitis in both animals and humans. Reference:
Campbell KL. Fatty acid supplementation and skin disease. Adv Clin Derm
1990;20(6):1475-1486.
Reference: Frithz A, Tollesson A. Essential fatty acids in preparations
for treatment of eczema. Sweden Patent No. 460762. Nov. 20, 1994
In lab tests it was found that artificially induced inflammation was
inhibited on mouse skin through the topical administration of borage
oil. Reference: Diezel WE, Schulz E, Shanks M, Heise H. Plant oils:
Topical application and anti-inflammatory effects (croton oil test).
Dermatol Monatsschr 1993;179:173
Elias has reported that a gel containing 1.5% borage
oil significantly reduced the TEWL of the skin of hairless mice maintained
on EFA deficient diets. Elias P, as quoted in Goldberg RL. Reference:
The Compounder's Corner: Exotic claims. Durg and Cosmetic Ind 1993;Jan:40
Infantile seborrhoeic dermatitis, a common condition in infants also
known as "cradle cap", has also been successfully treated
with borage oil. Reference: Tollesson A, Frithz A. Borage oil: An
effective new treatment for infantile seborrhoiec dermatitis. Br J Dermatol
1993;129:95
Deficiency of EFAs in many species is manifested early on as skin changes.
Skin becomes rough and inflamed with a poor wound healing capacity.
Just about any type of dermatitis can favorably respond to EFA supplementation.
Reference: Wright S. Essential fatty acids in clinical dermatology.
J Nutr Med 1:301-313, 1990.
Topical application along with oral supplementation may speed the effectiveness
of EFAs. Eczema, psoriasis and acne all have been shown to respond
to EFA supplementation. Reference: Wright S, Burton JL. Evening
primrose seed oil improves atopic eczema. Lancet ii:1120-1122,
1982.
Reference: Bittiner SB, Tucker WFG, Cartwright I, Bleehen SS.
A double blind randomized, placebo-controlled trial of fish oil in psoriasis.
Lancet i:378-380, 1988.
Reference: Downin DT, Stweart ME, Wertz PW, Strauss JS. Essential
fatty acids and acne. J Am Acad Dermat 14:221-4, 1986.
“Healthy skin depends on adequate amounts of
lipid, in particular certain polyunsaturated fatty acids called essential
fatty acids (EFAs), for moisture, suppleness and smoothness as well
as to prevent skin disorders. The most important polyunsaturated fatty
acids for maintenance of healthy skin and for the alleviation of skin
disorders are the essential fatty acids of the omega-6 family.”
Reference: Ziboh V, Miller C. Essential fatty acids and polyunsaturated
fatty acids: Significance in cutaneous biology. Annual Review of Nutrition.
1990;10:433
“Both oral and topical administration of GLA has been effective
in reducing these as well as reducing redness and erythema due to UV
radiation and improving healing of wounds.” Reference: Campbell
KL. Fatty acid supplementation and skin disease. Advanced Clinical Dermatology.
1990;20(6):1475-1486
“At least one study has shown that topical applications of EFAs
may also help control the damage caused by Ultraviolet-B.” Ando
H, Ryu A, Hashimot A, Oka M, Ichihashi M. Linoleic acid and alpha-linolenic
acid lightens ultraviolet-induced hyperpigmentation of the skin. Reference:
Archive of Dermatology Research. 1998;290:37538
“Topical application of oils rich in linoleic acid are highly
beneficial to the skin. Using volunteers suffering EFA deficiencies,
researchers found that topical application of sunflower seed oil (also
rich in EFAs) markedly increased the skin's linoleic acid levels; that
loss of water was highly decreased; and scaly lesions disappeared.”
Reference: C Prottey, PJ Hartop, and M Press. Correction of the
cutaneous manifestations of essential fatty acid deficiency in man by
application of sunflower-seed oil to the skin. Journal of Investigative
Dermatology. 1975 64: 228-234.
“Research with topical applications revealed Borage Oil’s
ability to provide the same level of improvement as it did when taken
orally. Even environmentally damaged and habitually dry skin received
renewed moisture and smoothness.” Reference: Gittleman, A.L.
GLA: The “good” omega-6. Total Health for Longevity Magazine.
September 2000.
“The properties contained in borage oil are so
notable in enhancing skin that both the Journal of American Academy
of Dermatology and the British Journal of Dermatology recognize its
benefits.” Reference: Gittleman, A.L. GLA: The “good”
omega-6. Total Health for Longevity Magazine. September 2000.
“EFAs actually increase cell resilience and moistens the fatty
layer beneath the skin.” Gittleman, A.L. GLA: The “good”
omega-6. Total Health for Longevity Magazine. September 2000.“Topical
application along with oral supplementation may speed the effectiveness
of EFAs. Eczema, psoriasis and acne all have been shown to respond to
EFA supplementation.” Wright S, Burton JL. Evening
primrose seed oil improves atopic eczema. Reference:
Lancet ii:1120-1122, 1982.; Bittiner SB, Tucker WFG, Cartwright
I, Bleehen SS. A double blind randomized, placebo-controlled trial
of fish oil in psoriasis. Reference: Lancet i:378-380, 1988.; and
Downin DT, Stweart ME, Wertz PW, Strauss JS. Essential fatty acids
and acne. J Am Acad Dermat 14:221-4, 1986.
Soy:
Soy was originally considered a waste product. It was used to rotate
crops. Reference:Katz, Solomon H., "Food and Biocultural Evolution:
A Model for the Investigation of Modern Nutritional Problems",
Nutritional Anthropology, Alan R. Liss Inc., 1987, p. 50.
Soy has never attained GRAS (Generally Recognized As Safe) status. Soy
protein did have approval for use as a binder in cardboard boxes, and
this approval was allowed to continue, as researchers considered that
migration of nitrites from the box into the food contents would be too
small to constitute a cancer risk. FDA officials called for safety specifications
and monitoring procedures before granting of GRAS status for food. These
were never performed for soy. To this day, use of soy protein is codified
as GRAS only for this limited industrial use as a cardboard binder.
This means that soy protein must be subject to pre-market approval procedures
each time manufacturers intend to use it as a food or add it to a food.
Reference:FDA ref 72/104, Report FDABF GRAS - 258.
Soy is one of the most processed and genetically modified foods on the
market. Soy also has one of the highest percentages of contamination.
A purée of cooked soybeans could be precipitated with calcium
sulfate or magnesium sulfate (plaster of Paris or Epsom salts) to make
a smooth, pale curd - tofu or bean curd. Reference: Cinderella’s
Dark Side, Sally Fallon & Mary G. Enig, Ph.D., Dec. 3, 2002.
Soy is a potent enzyme inhibitor. During digestion, soy inhibits trypsin,
which is a necessary digestive enzyme. These trypsin inhibitors are
large, tightly folded proteins that are not completely deactivated during
ordinary cooking. They can produce serious gastric distress, reduced
protein digestion and chronic deficiencies in amino acid uptake. In
test animals, diets high in trypsin inhibitors cause enlargement and
pathological conditions of the pancreas, including cancer. Reference:Rackis,
Joseph J. et al., "The USDA trypsin inhibitor study. I. Background,
objectives and procedural details", Qualification of Plant Foods
in Human Nutrition, vol. 35, 1985.
Soybeans are high in phytic acid, which can block the uptake of essential
minerals – calcium, magnesium, copper, iron, and especially zinc
(zinc is utilized by the body more than any other mineral). Reference:
Sally Fallon & Mary G. Enig, Ph.D.
Soy contains goitrogens – substances that depress thyroid function.
Soy based formula can cause thyroid problems in babies. Soy stunts the
growth and sexual development of male babies and children. The trypsin
inhibitors and harmogglutinin in soy are growth inhibitors. Females
who consumed soy milk as infants, have been shown to begin sexual development
as early as 3 years old. Other sexual complications may develop later
in life. References: Hagger, C. and J. Bachevalier, "Visual
habit formation in 3-month-old monkeys (Macaca mulatta): reversal of
sex difference following neonatal manipulations of androgen", Behavior
and Brain Research (1991) 45:57-63.
References: Ross, R.K. et al., "Effect of in-utero exposure to
diethylstilbestrol on age at onset of puberty and on post-pubertal hormone
levels in boys", Canadian Medical Association Journal 128(10):1197-8,
May 15, 1983.
Soybeans also contain haemagglutinin, a clot-promoting substance that
causes red blood cells to clump together. Reference: Cinderella’s
Dark Side, by Sally Fallon: & Mary G. Enig, Ph.D., Dec. 3, 2002.
Phytate reduction of zinc absorption has been demonstrated
in numerous studies. These results are summarized in Leviton, Richard,
Tofu, Tempeh, Miso and Other Soy foods: References: The 'Food of
the Future' - How to Enjoy Its Spectacular Health Benefits, Keats Publishing,
Inc., New Canaan, CT, USA, 1982, p. 1415.
Ross, R.K. et al., "Effect of in-utero exposure to diethylstilbestrol
on age at onset of puberty and on post-pubertal hormone levels in boys",
References: Canadian Medical Association Journal 128(10):1197-8,
May 15, 1983.
Soy and Infants:
It is estimated that an infant exclusively fed soy
formula receives the estrogenic equivalent (based of body weight) of
at least 5 birth control pills per day. Reference: Irvine, C. et
al., "The Potential Adverse Effects of Soybean Phytoestrogens in
Infant Feeding", New Zealand Medical Journal May 24, 1995, p. 318.
…18% higher incidence in autoimmune thyroid disease
in infants who are fed soy
formula. Reference: J Am Coll Nutr 1990, Apr; 9(2): 164-167
[Dietary fat] is a required nutrient for an infant's brain and nerve
development. Compared to breast-fed infants, infants who were fed hydrolyzed
soy (processed) protein showed significant reduced growth in weight
and length, as well as total blood protein Reference: Acta Paediatr
Suppl, Sept. 1994; 402: 100-104, and Eur J Clin Nutr, Sept. 1995; 49
Suppl 1: S26-38
Soy-based infant products often contain double the amount of protein
supplied by mother's milk. (This is not good - the baby is supposed
to get fats, not excessive protein.) Soy formula is clearly not a proper
"substitute" Reference: Adv Exp Med Biol, 1991;
289: 389-402
Soy and Sex:
Celibate monks living in monasteries and leading a vegetarian lifestyle
find soy foods quite helpful because they dampen libido. Reference:
Cinderella’s Dark Side, Sally Fallon & Mary G. Enig, Ph.D.,
Dec. 3, 2002.
Sugar: (see Carbohydrates)
High sugar intake, excessive insulin production and excess glucose in
the body can react with proteins in a process called glycation. These
sugar damaged proteins cause premature ageing of many tissues (including
collagen in the skin) and lead to joint problems, loss of energy, stiffness,
difficulties in weight control and many other unwelcome problems. Reference:
New York Academy of Sciences, BPDG Meeting, January 29, 2002, “AGEing
- Non-Enzymatic Glycation and Carbonyl Stress,”Organizers: Marla
Weetall, Ph.D., Novartis Institute for Biomedical Research: Robert deGroof,
Ph.D., Alteon Inc.
8. Hospital patients not allowed more than 7 tsp sugar
per hour; Americans told by U.S. government [food pyramid] and nutritionists
to eat up to 20 tsp sugar at each meal: breakfast, lunch, and dinner!
Reference: Body Fluids and Electrolytes, pgs: 71-72.
Fructose (fruit sugar):
Humans can’t digest the fructose of more than 2 pieces of fruit
a day: Reference: Basic Medical Biochemistry: A Clinical Approach,
pg. 404. Dawn B. Marks, Allan D. Marks, Colleen M. Smith, Lippincott,
Williams & Wilkins, August, 1996, ISBN: 068305595X
Sweeteners:
Methanol(and other ingredients ending in “nol”) = wood alcohol
– very dangerous poison to the human body (The main ingredient
of Aspartame).
The lists of reported negative effects from consumption
of aspartame are too many and too long to include here. A good source
for this information currently is: http://www.holisticmed.com/aspartame/suffer.faq.
Or request a copy of the paper (including references), Reference:
“Reported Aspartame Toxicity Effects”, from the web site
listed above.
Areas reported to be effected by Aspartame consumption:
Eye
Ear
Neurological
Psychologic-Psychiatric
Chest
Gastrointestinal
Skin and Allergies
Endocrine and Metabolic
And more…
Reports in scientific literature of aspartame-caused toxicity reactions:
Blumenthal (1997), Drake (1986), Johns (1986), Lipton (1989), McCauliffe
(1991), Novick (1985), Watts (1991), Walton (1986, 1988), and Wurtman
(1985).
Note: (Nutra-Sweet + MSG = Brain damage in children = Behavior disorders
= crime= perceived control necessity = totalitarian surveillance and
control…) Reference: A study by Dr. John Olney, professor
of neuropathology and psychiatry, Washington School of Medicine, St.
Louis, Missouri.
Sugar alcohol (also known as polyols) comes from fruit. They are used
as sweeteners and bulking agents in processed foods. They provide _
to 1/3 less calories than sugar.
Sugar Alcohols:
Mannitol Sorbitol Xylitol
Lactitol Isomalt
Maltitol Hydrogenated Starch Hydrolysates (HSH)
Negative effects of sugar alcohol consumption (in large
amounts):
Bloating
Diarrhea
Weight gain
Can raise blood sugar
Reference: Yale-New Haven Hospital, 20 York St., New Haven CT 06510-3202,
1999-2002.
Sucralose (Sold as Splenda™):
Sucralose is produced by chlorinating sugar (sucrose). This involves
chemically changing the structure of the sugar molecules by substituting
three chlorine atoms for three hydroxyl groups.
Possible negative effects of Sucralose (results from controlled
studies on rats):
Shrunken thymus glands (up to 40% shrinkage)
Enlarged liver and kidneys.
Atrophy of lymph follicles in the spleen and thymus
Increased cecal weight
Reduced growth rate
Decreased red blood cell count
Hyperplasia of the pelvis
Extension of the pregnancy period
Aborted pregnancy
Decreased fetal body weights and placental weights
Diarrhea
[Toxicologist Judith] Bellin reviewed studies on rats starved under
experimental conditions, and concluded that their growth rate could
be reduced by as much as a third without the thymus losing a significant
amount of weight (less than 7 percent). The changes were much more marked
in rats fed on sucralose. While the animals' growth rate was reduced
by between 7 and 20 percent, their thymuses shrank by as much as 40
percent. Reference: New Scientist 23 Nov 1991, pg 13.
Syndrome X:
- Central obesity (excessive fat tissue in the abdominal region –
spare tire).
- Glucose intolerance.
- Hyperlipidemia -- primarily high triglycerides and low HDL cholesterol.
- High blood pressure.
Syndrome X is not a new disease, but a new term for a cluster of symptoms
that can include: insulin resistance (the inability to properly deal
with dietary carbohydrates and sugars), abnormal blood fats (such as
elevated triglycerides), obesity, and high blood pressure. Reference:
Syndrome X, The Complete Nutritional Program to Prevent and Reverse
Insulin Resistance, Jack Challem, The Nutrition Reporter™, Burt
Berkson, M.D., Ph.D., Melissa Diane Smith, nutrition counselor, Publication
Date: January 14, 2000